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control igg antibody  (R&D Systems)


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    R&D Systems control igg antibody
    Control Igg Antibody, supplied by R&D Systems, used in various techniques. Bioz Stars score: 93/100, based on 24 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mouse+ccl7+mcp+3+antibody/Mouse+CCL7%2FMCP-3%2FMARC+Antibody/bio_rxiv__64898__2026__04__21__720007-200-17-13
    Average 93 stars, based on 24 article reviews
    control igg antibody - by Bioz Stars, 2026-10
    93/100 stars

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    Related Articles

    Neutralization:

    Article Title: Bone marrow B cell collapse promotes bone metastasis in breast cancer
    Article Snippet: For CXCR4 inhibition, PBS vehicle or 10 mg/kg AMD3100 (Abcam, AB120718) were injected daily. .. For CCL7/MCP-3 neutralization, mice were injected with 20 μg of mouse CCL7/MCP-3 antibody (R&D systems, AF-456-NA) or control IgG antibody every 3 days. .. Animals were anesthetized with 2% isoflurane and 2 x 10 5 E0771 cells or 1 x 10 5 4T1.2 cells resuspended in 100 μL of PBS were injected into the left cardiac ventricle using a 31 G syringe needle (BD, 328438).

    Injection:

    Article Title: Bone marrow B cell collapse promotes bone metastasis in breast cancer
    Article Snippet: For CXCR4 inhibition, PBS vehicle or 10 mg/kg AMD3100 (Abcam, AB120718) were injected daily. .. For CCL7/MCP-3 neutralization, mice were injected with 20 μg of mouse CCL7/MCP-3 antibody (R&D systems, AF-456-NA) or control IgG antibody every 3 days. .. Animals were anesthetized with 2% isoflurane and 2 x 10 5 E0771 cells or 1 x 10 5 4T1.2 cells resuspended in 100 μL of PBS were injected into the left cardiac ventricle using a 31 G syringe needle (BD, 328438).

    Control:

    Article Title: Bone marrow B cell collapse promotes bone metastasis in breast cancer
    Article Snippet: For CXCR4 inhibition, PBS vehicle or 10 mg/kg AMD3100 (Abcam, AB120718) were injected daily. .. For CCL7/MCP-3 neutralization, mice were injected with 20 μg of mouse CCL7/MCP-3 antibody (R&D systems, AF-456-NA) or control IgG antibody every 3 days. .. Animals were anesthetized with 2% isoflurane and 2 x 10 5 E0771 cells or 1 x 10 5 4T1.2 cells resuspended in 100 μL of PBS were injected into the left cardiac ventricle using a 31 G syringe needle (BD, 328438).



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    Figure 4: Genetic and pharmacologic blockade of CCR2-CCL2 axis protects from hypoxic PH. (A) Schematic showing the BM reconstitution of Ccr2-/- and WT BM into lethally irradiated wildtype mice. Wildtype mice reconstituted with Ccr2-/- BM were protected from hypoxic PH by attenuated (B) RVSP (N=7-11/group) and (C) RV hypertrophy (N=7-11/group) as measured by Fulton Index, compared to wildtype mice that were reconstituted with wildtype BM. (D) Schematic showing pharmacological blockade of CCR2 ligands CCL2 or <t>CCL7</t> using anti-CCL2 or anti-CCL7 neutralizing antibody treatment. Hypoxia exposed wildtype mice treated with CCL2 NAb but not CCL7 NAb showed lower (E) RVSP (N=6/group) and (F) RV hypertrophy (N=6/group). TSP-1 levels in (G) lungs (N=6/group) and (H) blood (N=6/group); and TGF-β1 levels in (I) lungs (N=6/group) and (J) blood (N=6/group) compared to wildtype mice treated with isotype control antibody. Data in all panels followed a normal distribution. ANOVA with the Tukey test was performed for multiple comparisons. Data were obtained from the female mice. mean ± SD
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    Image Search Results


    Figure 4: Genetic and pharmacologic blockade of CCR2-CCL2 axis protects from hypoxic PH. (A) Schematic showing the BM reconstitution of Ccr2-/- and WT BM into lethally irradiated wildtype mice. Wildtype mice reconstituted with Ccr2-/- BM were protected from hypoxic PH by attenuated (B) RVSP (N=7-11/group) and (C) RV hypertrophy (N=7-11/group) as measured by Fulton Index, compared to wildtype mice that were reconstituted with wildtype BM. (D) Schematic showing pharmacological blockade of CCR2 ligands CCL2 or CCL7 using anti-CCL2 or anti-CCL7 neutralizing antibody treatment. Hypoxia exposed wildtype mice treated with CCL2 NAb but not CCL7 NAb showed lower (E) RVSP (N=6/group) and (F) RV hypertrophy (N=6/group). TSP-1 levels in (G) lungs (N=6/group) and (H) blood (N=6/group); and TGF-β1 levels in (I) lungs (N=6/group) and (J) blood (N=6/group) compared to wildtype mice treated with isotype control antibody. Data in all panels followed a normal distribution. ANOVA with the Tukey test was performed for multiple comparisons. Data were obtained from the female mice. mean ± SD

    Journal: Journal of Clinical Investigation

    Article Title: Monocytes and interstitial macrophages contribute to hypoxic pulmonary hypertension

    doi: 10.1172/jci176865

    Figure Lengend Snippet: Figure 4: Genetic and pharmacologic blockade of CCR2-CCL2 axis protects from hypoxic PH. (A) Schematic showing the BM reconstitution of Ccr2-/- and WT BM into lethally irradiated wildtype mice. Wildtype mice reconstituted with Ccr2-/- BM were protected from hypoxic PH by attenuated (B) RVSP (N=7-11/group) and (C) RV hypertrophy (N=7-11/group) as measured by Fulton Index, compared to wildtype mice that were reconstituted with wildtype BM. (D) Schematic showing pharmacological blockade of CCR2 ligands CCL2 or CCL7 using anti-CCL2 or anti-CCL7 neutralizing antibody treatment. Hypoxia exposed wildtype mice treated with CCL2 NAb but not CCL7 NAb showed lower (E) RVSP (N=6/group) and (F) RV hypertrophy (N=6/group). TSP-1 levels in (G) lungs (N=6/group) and (H) blood (N=6/group); and TGF-β1 levels in (I) lungs (N=6/group) and (J) blood (N=6/group) compared to wildtype mice treated with isotype control antibody. Data in all panels followed a normal distribution. ANOVA with the Tukey test was performed for multiple comparisons. Data were obtained from the female mice. mean ± SD

    Article Snippet: Neutralizing Antibody and Pharmacological Treatment: Neutralizing mouse antibodies against CCL2 (Clone: 2H5; Cat.# BE0185), CCL7 (R&D System; Cat.# AF-456-NA), and isotype control (Cat.# BE0091; BioXCell, West Lebanon, NH, USA) were reconstituted in phosphatebuffered saline (PBS).

    Techniques: Irradiation, Control